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ROZUMIEM

How does kamiwaza work?

Many factors, such as nutrition, food and environmental pollution (air, water), side effects of drugs, type of work, mental stress, trigger excessive amounts of oxygen free radicals in the cells, leading to the so-called oxidative stress. Stress, regardless of the type of factor that caused it, leads to an excessive production of free radicals that damage proteins and cause cell death. Reactive oxygen weakens the network of structural proteins, the so-called The cell "skeleton" (see Figure 1 - damaged cell). Damage to the hair follicle cells is the direct cause of pathological hair loss and alopecia. Without recognizing this mechanism, most of the hair regrowth stimulants used so far were haphazard and ineffective.

The main question was, how can this negative influence of stress factors be limited or neutralized? After examining hundreds of natural compounds derived from medicinal plants, substances were isolated that significantly increased the activity of several important anti-stress enzymes in the hair follicle cells, mainly the SOD enzyme, which by neutralizing the excess of reactive oxygen prevents stress damage. They also isolated substances that strongly increased the activity of chaperones that repair stress-damaged enzyme, structural and other proteins, thus restoring health to cells and hair (see Figure 2 - "repaired" cell). The results of these studies provided the basis for the development of a formula for the natural biostimulator KAMIWAZA.

Fig. 1 Diagram of a fragment of a cell damaged by stress 1) lipid membranes damaged by stress 2) Proteins - receptors responding to signals from outside the cell (e.g. hormones): inactive due to changing their shape and displacement after damage to structural proteins 3) Other proteins transmitting signals from outside the cell: inactive due to the change in their shape and displacement after damage to structural proteins 4) Complex of proteins carrying out biochemical reactions: inactive due to changing the shape of the reacting domains and lack of interaction proteins -receptors, protein complexes, various cell structures (so-called organelles), in the right places in the cell; damaged by stress.

Fig. 1 Diagram of a fragment of a cell damaged by stress 1) lipid membranes damaged by stress 2) Proteins - receptors responding to signals from outside the cell (e.g. hormones): inactive due to changing their shape and displacement after damage to structural proteins 3) Other proteins transmitting signals from outside the cell: inactive due to the change in their shape and displacement after damage to structural proteins 4) Complex of proteins carrying out biochemical reactions: inactive due to changing the shape of the reacting domains and lack of interaction proteins -receptors, protein complexes, various cell structures (so-called organelles), in the right places in the cell; damaged by stress.

contact

Ayla Biolabs,
Pogodna 20/3
05-077 Warsaw

503-156-928

kamiwazabio@gmail.com